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Publication Listing for the MeSH term ovarian. Found 9 abstracts

Behbakht K, Sill MW, Darcy KM, Rubin SC, Mannel RS, Waggoner S, Schilder RJ, Cai KQ, Godwin AK, Alpaugh RK. Phase II trial of the mTOR inhibitor, temsirolimus and evaluation of circulating tumor cells and tumor biomarkers in persistent and recurrent epithelial ovarian and primary peritoneal malignancies: A Gynecologic Oncology Group study. Gynecologic Oncology. 2011 Oct;123(1):19-26.   PMCID: PMC3336961 [Available on 2012/10/1]
Sasaroli D, Gimotty PA, Pathak HB, Hammond R, Kougioumtzidou E, Katsaros D, Buckanovich R, Devarajan K, Sandaltzopoulos R, Godwin AK, Scholler N, Coukos G, Stellani P VP. Novel surface targets and serum biomarkers from the ovarian cancer vasculature. Cancer Biology & Therapy. 2011 Aug;12(3):169-80.   PMCID: PMC3230481
Apicella C, Dowty JG, Dite GS, Jenkins MA, Senle RT, Daly MB, Andrulis IL, John EM, Buys SS, Li FP, Glendon G, Chung W, Ozcelik H, Miron A, Kotar K, Southey MC, Foulkes WD, Hopper JL. Validation study of the LAMBDA model for predicting the BRCA1 or BRCA2 mutation carrier status of North American Ashkenazi Jewish women. Clinical genetics. 2007 Aug;72(2):87-97.
Bast RC, Thigpen JT, Arbuck SG, Basen-Engquist K, Burke LB, Freedman R, Horning SJ, Ozols R, Rustin GJ, Spriggs D, Wenzel LB, Pazdur R. Clinical trial endpoints in ovarian cancer: Report of an FDA/ASCO/AACR public workshop. Gynecologic Oncology. 2007 Nov;107(2):173-6.
Cai KQ, Klein-Szanto A, Karthik D, Edelson M, Daly MB, Ozols RF, Lynch HT, Godwin AK, Xu XX. Age-dependent morphological alterations of human ovaries from populations with and without BRCA mutations. Gynecologic Oncology. 2006 Nov;103(2):719-28.
Disis ML, Rivkin SE, Baron A, Markman M, Connolly D, Ueland F, Kohn E, Trimble E, Berek JS. Progress in ovarian cancer research: Proceedings of the 5th biennial ovarian cancer research symposium. INTERNATIONAL JOURNAL OF GYNECOLOGICAL CANCER. 2006 Mar;16(2):463-9.
Diefenbach MA, Miller SM, Daly MB. Specific worry about breast cancer predicts mammography use in women at risk for breast and ovarian cancer. Health Psychology. 1999 Sep;18(5):532-6.
Ruggeri BA, Huang LY, Wood M, Cheng JQ, Testa JR. Amplification and overexpression of the AKT2 oncogene in a subset of human pancreatic ductal adenocarcinomas. Molecular Carcinogenesis. 1998 Feb;21(2):81-6.
Altomare DA, Kozak CA, Sonoda G, Testa JR. Chromosome mapping of the mouse Akt2 gene and Akt2 pseudogene. Cytogenetics and Cell Genetics. 1996 Jan;74(4):248-51.
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MeSH cloud from publications including the MeSH term ovarian

ovarian cancer expression carcinomas breast cancer c-akt BRCA2 gene-expression carcinoma serum BRCA1 research morphological changes are associated with BRCA 66 ovaries were from surgeries due to non-ovarian-related AKT cancer (c) 2006 Elsevier Inc All rights reserved trial collection of ovaries from prophylactic oophorectomies and control inhibitor mortality events protein-kinase-b signaling proteins CA-125 aging relate to BRCA1 epithelium metastatic colorectal-cancer mammalian target activation therapeutic target RISK adherence cancer worry OVARIAN-CANCER and gonadotropin stimulation produces ovarian morphological changes SUSCEPTIBILITY GENES menopause JEWS Ashkenazi Jewish angiogenesis therapy WOMEN and morphological changes including ovarian tissues was examined mutations immunohistochemistry progression diagnostics ovulation PHENOTYPE vessels common-sense platinum-resistant non-neoplastic diseases to determine if ovarian morphological changes Results No statistically significant difference in frequency of these SURFACE breast-cancer with BRCA1 localization BRCA2 genotypes or are Methods We assembled a panel of archived ovarian tissues: 52 ovarian age refractory pancreatic cancer especially inclusion cysts features-may associate with age or receptors invaginations papillomatosis-inclusion cysts-and epithelial pre-malignant lesion tissue blocks were from prophylactic oophorectomies of a high-risk (HR) Conclusions This study suggests that no significant increase in the contribute presence of non-neoplastic ovarian morphological changes is associated UNITED-STATES etiological factor we have investigated a recent cancer precursors is controversial Here Ovarian clinical trial resistant prostate-cancer FDA women age 45-54 of either HR or NR groups the frequency of these histological BRCA2 mutations Rather PI3K pathway population proteins information ca125 MICE fluorescence insitu hybridization bevacizumab FAMILY-HISTORY stratification were assessed in a blinded fashion histolopathologic features was found between HR and NR groups However PROPHYLACTIC OOPHORECTOMY SPECIMENS kinase CARRIERS inclusion cysts and deep invaginations were found much more commonly in translational profiling Circulating tumor cells markers biomarkers family history mTOR disease prophylactic oophorectomies PREVALENCE diagnosis CA125 2 genotypes or reproductive history BREAST-CANCER PATIENTS CANCER-RISK risk women 185DELAG carcinoma patients referred to as normal-risk (NR) group The morphology of diseases Oncology OVULATION COMMON BRCA1 oncogene model tumor inhibition mammography adherence gene amplification TUMORIGENESIS metastasis GONADOTROPINS AB Objective From analysis of pre-cancer ovarian tissues obtained from ovaries from surgeries due to other non-ovarian-related cancer or self-examination vascular peripheral-blood endpoints these histological features may promote the transformation of genetically compromised epithelial cells in the development of ovarian dna endothelial-cells menopausal status We propose that ovulatory and perimenopausal Obstetrics & Gynecology several studies reported the increased prophylactic oophorectomies identification but whether these ovarian morphological changes in high-risk ovaries AKT2 oncogene
Last updated on Wednesday, March 04, 2020