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Publication Listing for the MeSH term epithelium. Found 8 abstracts

Whelan KA, Godwin BC, Wilkins B, Elci OU, Benitez A, DeMarshall M, Sharma M, Gross J, Klein-Szanto AJ, Liacouras CA, Dellon ES, Spergel JM, Falk GW, Muir AB, Nakagawa H. Persistent Basal Cell Hyperplasia is Associated with Clinical and Endoscopic Findings in Patients With Histologically Inactive Eosinophilic Esophagitis. Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association. 2019 Sep 06;.
Kotredes KP, Thomas B, Gamero AM. The Protective Role of Type I Interferons in the Gastrointestinal Tract. Frontiers in immunology. 2017 Apr;8:410.   PMCID: PMC5382159
Bendoraite A, Knouf EC, Garg KS, Parkin RK, Kroh EM, O'Briant KC, Ventura AP, Godwin AK, Karlan BY, Drescher CW, Urban N, Knudsen BS, Tewari M. Regulation of miR-200 family microRNAs and ZEB transcription factors in ovarian cancer: Evidence supporting a mesothelial-to-epithelial transition. Gynecologic Oncology. 2010 Jan;116(1):117-25.   PMCID: PMCID: PMC2867670 [Available on 2011/1/1]
Ohashi S, Natsuizaka M, Yashiro-Ohtani Y, Kalman RA, Nakagawa M, Wu LZ, Klein-Szanto AJ, Herlyn M, Diehl JA, Katz JP, Pear WS, Seykora JT, Nakagawa H. NOTCH1 and NOTCH3 Coordinate Esophageal Squamous Differentiation Through a CSL-Dependent Transcriptional Network. Gastroenterology. 2010 Dec;139(6):2113-23.   PMCID: PMC2997138
Cai KQ, Yang WL, Capo-Chichi CD, Vanderveer L, Wu H, Godwin AK, Xu XX. Prominent expression of metalloproteinases in early stages of ovarian turnorigenesis. Molecular Carcinogenesis. 2007 Feb;46(2):130-43.
Cai KQ, Klein-Szanto A, Karthik D, Edelson M, Daly MB, Ozols RF, Lynch HT, Godwin AK, Xu XX. Age-dependent morphological alterations of human ovaries from populations with and without BRCA mutations. Gynecologic Oncology. 2006 Nov;103(2):719-28.
Beauparlant SL, Read PW, Di Cristofano A. In vivo adenovirus-mediated gene transduction into mouse endometrial glands: a novel tool to model endometrial cancer in the mouse. Gynecologic Oncology. 2004 Sep;94(3):713-8.
Roland IH, Yang WL, Yang DH, Daly MB, Ozols RF, Hamilton TC, Lynch HT, Godwin AK, Xu XX. Loss of surface and cyst epithelial basement membranes and preneoplastic morphologic changes in prophylactic oophorectomies. Cancer. 2003 Dec;98(12):2607-23.
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MeSH cloud from publications including the MeSH term epithelium

epithelium expression CANCER-RISK inflammation OVULATION prophylactic oophorectomies MicroRNA these histological features may promote the transformation of disabled-2 genetically compromised epithelial cells in the development of ovarian menopausal status We propose that ovulatory and perimenopausal several studies reported the increased prophylactic oophorectomies miR-200 MAMMALIAN OVULATION identification but whether these ovarian morphological changes in high-risk ovaries TUMOR DISABLED-2 morphological changes are associated with BRCA 66 ovaries were from surgeries due to non-ovarian-related collagen IV cancer (c) 2006 Elsevier Inc All rights reserved GROWTH-REGULATION collection of ovaries from prophylactic oophorectomies and control esophagus outcome aging relate to BRCA1 mice PROPHYLACTIC OOPHORECTOMIES activation CYCLOOXYGENASE-2 COX-2 premalignant lesion laminin EXTRACELLULAR-MATRIX OVARIAN-CANCER cells ovarian and gonadotropin stimulation produces ovarian morphological changes intestine menopause cell-proliferation WOMEN ovarian carcinoma and morphological changes including ovarian tissues was examined receptor adenoviral vectors pten double-negative feedback interferon progression ovulation mouse models ZEB2 BRCA2 non-neoplastic diseases to determine if ovarian morphological changes Results No statistically significant difference in frequency of these SURFACE GONADOTROPINS AB Objective From analysis of pre-cancer ovarian tissues obtained from microbiome growth-factor receptor Ovarian cancer MMPs prognostic factor TRANSGENIC MICE with BRCA1 BRCA2 genotypes or are Methods We assembled a panel of archived ovarian tissues: 52 ovarian mesenchymal transition especially inclusion cysts features-may associate with age or IN-VIVO invaginations papillomatosis-inclusion cysts-and epithelial pre-malignant lesion tissue blocks were from prophylactic oophorectomies of a high-risk (HR) repressors zeb1 Conclusions This study suggests that no significant increase in the TUMORIGENICITY presence of non-neoplastic ovarian morphological changes is associated KeyWords Plus: EXPRESSION etiological factor we have investigated a recent cancer precursors is controversial Here membrane SPONTANEOUS TRANSFORMATION women age 45-54 of either HR or NR groups the frequency of these histological BRCA2 mutations Rather PROSTAGLANDIN E-2 population basement membrane cancer MICE CELLS Cox-2 FAMILY-HISTORY stratification were assessed in a blinded fashion histolopathologic features was found between HR and NR groups However Feedback loop carcinoma PROPHYLACTIC OOPHORECTOMY SPECIMENS CARRIERS inclusion cysts and deep invaginations were found much more commonly in e-cadherin conditional knockout BRCA1 endometrial cancer delta-ef1 PROGRESSION ovarian cancer 2 genotypes or reproductive history keratinocytes surface commitment Mesothelial PTEN suppression t-cell TUMORIGENESIS NOTCH3 NOTCH1-Esophageal Epithelium-Squamous Differentiation MATRIX METALLOPROTEINASES ovaries from surgeries due to other non-ovarian-related cancer or skin SURFACE EPITHELIAL-CELLS referred to as normal-risk (NR) group The morphology of diseases basement preneoplastic changes carcinogenesis
Last updated on Wednesday, February 05, 2020